Pseudomonas syringae (P.syringae) uses a typeIII secretion system (T3SS) called the Hrp T3SS to inject type III effectors into plant cells and suppress plant immune responses. Translocated type III effectors carry out specific functions within the host cell and collectively are required for P. syringae to cause disease. P.syringae pv. tomato DC3000 injects more than 30 effector proteins into plant cells. Among those HopB1 that is secreted in culture and translocated into plant cells via the T3SS. our data indicated that HopE1 targets MAP65 proteins for virulence revealing a novel mechanism for bacterial pathogenesis. My research goals are that study of those effectors to find out their roles on plant innate immunity. Besides effectors, part of my researches is about RNA binding protein, GRP7. Our research demonstrates that GRP7 has a broad effect on plant innate immunity and is a component of SA-dependent immune responses. I am trying to figure out RNA binding motif via RNA immunoprecipitation (RIP) sequencing that may explain more about the further process of GRP7 in Ararbidopsis.